Medical Imaging International
3T MRI Scanner Promises Improved Performance and Fewer Rescans eaturing advanced system architecture and a more powerful magnet, a newly-introduced 3T Magnetic Resonance Imaging (MRI) scanner can help clinicians personalize imaging scans and reduce the number of rescans, while improving scheduling and consistency. The MAGNETOM Vida scanner was developed by Siemens Healthineers (Erlangen, Germany; www.health care.siemens.com), and features Siemens’ BioMatrix, GO, Select&GO, and true 3T productivity technologies, and free-breathing, high-resolution cardiac MRI with compressed sensing cardiac cine. Siemens’ GO technologies enable a faster exam workflow, and higher throughput by facilitating workflow optimization. The front of the scanner has BioMatrix Select&GO touch interfaces that enable one-touch patient positioning for multiple regions, as well as an intelligent body model that saves repositioning time, and speeds up adjustments. The Siemens’ Dot workflow helps ensure consistent, high-quality imaging using automation features such as AutoAlign, AutoCoverage and AutoFoV. Inline image reconstructions are automated and use the Recon&GO functionality that allows automatic
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Cover Image: Courtesy of Philips Healthcare Dan Gueron Publisher Andrew Deutsch News Editor Joseph Ciprut Assistant Editor Brenda Silva New Products Editor Theresa Herman Regional Director Dr. Jutta Ciolek Regional Director Parker Xu Regional Director Katsuhiro Ishii Regional Director Hazel Tapia Reader Service Manager Arda Turac Production Director
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reconstructions to be carried out in the background. The MAGNETOM Vida also features a new viewing and reading solution called MR View&GO, that enables rapid quality checks and results distribution, following image acquisition and image reconstruction. Image: The MAGNETOM Vida MRI Scanner (Photo courtesy of Siemens Healthineers).
PET Radiotracer Identifies Atherosclerotic Inflammation new study describes a novel positron emission tomography (PET) radiotracer that can quickly and non-invasively identify life-threatening atherosclerotic plaques. Developed by researchers at the Munich Technical University (TUM; Germany; www.tum.de) and Klinikum rechts der Isar (Munich, Germany; www.mri.tum.de), gallium-68 (Ga-68)-pentixafor binds with nanomolar affinity to the CXC chemokine receptor type 4 (CXCR4, also known as fusin), which is present on the surface of inflammatory cells in atherosclerotic plaques. For the study, the researchers imaged seven atherosclerotic rabbits and five controls in vivo on a PET-MRI system after injection of 15 MBq/kg of 68Ga-pentixafor. One hour later, strong signals were detected with PET-MRI in atherosclerotic plaques of the abdominal aorta and right carotid artery, as compared to normal control arteries. The uptake in the vessel wall on radiographies was located in the macrophage-rich regions of atherosclerotic plaques, and correlated with the intensity of CXCR4 expression. The researchers also confirmed (with a small number of human patients) that the radiotracer detect-
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ed atherosclerotic plaques located in their carotid arteries. The study was published in the March 2017 issue of The Journal of Nuclear Medicine. “Ga-68-pentixafor binds more specifically to inflammatory cells than F-18-fluorodeoxyglucose and does not require the patient to fast for six hours before imaging,” said lead author Fabien Hyafil, MD, PhD, of Klinikum Rechts der Isar. “This new radiotracer will strongly facilitate the imaging of inflammation in atherosclerotic plaques with PET, and hopefully support the early detection and treatment of atherosclerosis, thus preventing heart attack or stroke.” Atherosclerotic plaques fall into two broad categories – stable and unstable. While stable atherosclerotic plaques, which tend to be asymptomatic, are rich in extracellular matrix (ECM) and smooth muscle cells, unstable plaques are rich in macrophages and foam cells, and the ECM separating the lesion from the arterial lumen is usually weak and prone to rupture, causing thromboembolism. In the extreme, the emboli can cause myocardial infarction (MI) when lodged in cardiac arteries, or a stroke when reaching the brain.
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ISSN 1068-1779 Vol.27 No.3. Published, under license, by Globetech Media, LLC. Copyright © 2017. All rights reserved. Reproduction in any form is forbidden without express permission.
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Medical Imaging International August-September/2017
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