A Guide to Necropsy Diagnosis in Swine Pathology

Marcelo De las Heras Guillamón José Antonio García de Jalón

A guide to necropsy diagnosis in swine pathology

AGuidetoNecropsy_Cubierta_SERVET.indd 1

A guide to necropsy diagnosis in swine pathology

Marcelo De las Heras Guillamón - José Antonio García de Jalón

26/06/14 18:48

THE VETERINARY PUBLISHING COMPANY PRODUCTION ANIMALS

SWINE

A Guide to Necropsy Diagnosis in Swine Pathology

Aimed at veterinary surgeons, students, teachers and other professionals in the veterinary sector

Authors: Marcelo De las Heras and José Antonio García de Jalón

Format: 17 x 24 cm Number of pages: 184 Number of pictures: 250 Binding: Hardcover ISBN: 978-84-92569-76-2 Editorial: Servet PVP: 35 e

In this book we first propose the correct way to make a quick and complete necropsy which reveals the different organs and structures, offering a technique that can provide us with as much information as possible. Secondly, and directly connected with the necropsy process and maintaining both the orders and the observation plane, we show a significant number of lesions with their respective interpretations and possible relationships with one or more diseases.

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A Guide to Necropsy Diagnosis in Swine Pathology TABLE OF CONTENTS 1. Carcass opening technique and observation planes 2. External appearance: cutaneous plane, hooves, snout and natural openings 3. Plane of subcutaneous tissue and musculoskeletal system 4. Plane of neck and thorax 5. Abdominal plane: digestive tube 6. Abdominal plane: liver, spleen, kidneys and genitourinary system 7. Head: skull and nasal cavities

Centro Empresarial El Trovador, planta 8, oficina I - Plaza Antonio Beltrán Martínez, 1 • 50002 Zaragoza - España Tel.: 976 461 480 • Fax: 976 423 000 • pedidos@grupoasis.com • Grupo Asís Biomedia, S.L.

2.16 Growing pig showing recumbency, lethargy and fever. May be caused by classical swine fever (Hog cholera). Similar clinical signs can be observed in Aujeszky´s disease and in streptococcosis.

2.18 Growing pig. Ulcers and erosions in the snout area. This may be due to Swine vesicular disease (SVD) which also presents vesicles which are easily ruptured leaving erosions or ulcers.

2.19 Growing pig. Erosions in the snout area following the formation of vesicles.

Another example of Swine vesicular disease (SVD).

2.17 Weaned piglet. Porcine atrophic rhinitis. Note shortening and abnormality of the snout as a consequence of atrophic and chronic catarrhal inflammation.

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2.16 Growing pig showing recumbency, lethargy and fever. May be caused by classical swine fever (Hog cholera). Similar clinical signs can be observed in Aujeszky´s disease and in streptococcosis.

2.18 Growing pig. Ulcers and erosions in the snout area. This may be due to Swine vesicular disease (SVD) which also presents vesicles which are easily ruptured leaving erosions or ulcers.

2.19 Growing pig. Erosions in the snout area following the formation of vesicles.

Another example of Swine vesicular disease (SVD).

2.17 Weaned piglet. Porcine atrophic rhinitis. Note shortening and abnormality of the snout as a consequence of atrophic and chronic catarrhal inflammation.

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4.5 Sow. Subepicardial hemorrhages related with bacterial septicemia in the course of pyometra.

4.4 Weaned piglet. Subepicardial hemorrhages in the course of acute classical swine fever (Hog cholera). Could also be associated with other septicemic conditions like septicemic Salmonellosis.

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4.6 Weaned piglet. Edema and subepicardial hemorrhage due to dietetic microangiopathy or vitamin E/selenium deficiency (Mulberry heart disease).

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4.5 Sow. Subepicardial hemorrhages related with bacterial septicemia in the course of pyometra.

4.4 Weaned piglet. Subepicardial hemorrhages in the course of acute classical swine fever (Hog cholera). Could also be associated with other septicemic conditions like septicemic Salmonellosis.

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4.6 Weaned piglet. Edema and subepicardial hemorrhage due to dietetic microangiopathy or vitamin E/selenium deficiency (Mulberry heart disease).

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5.57 Starter pig with severe necrotic-diphteroid typhlitis. Note necrotic appearance of mucosa and abundant layer of fibrin. Salmonellosis.

5.58 Finishing pig with severe distension of intestinal loops due to fecal retention.

The animal presented pendulous abdomen and abdominal dilatation due to rectal stenosis.

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5.59 Rectal stenosis in a growing pig. Note intense narrowing of the lumen due to sclerosis of the intestinal wall. Usually associated with ulcerative proctitis and thrombosis of hemorrhoid vessels as sequel of salmonellosis. However, stenosis is also observed in rectal prolapses without antecedents of enterocolitis by Salmonella.

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5.57 Starter pig with severe necrotic-diphteroid typhlitis. Note necrotic appearance of mucosa and abundant layer of fibrin. Salmonellosis.

5.58 Finishing pig with severe distension of intestinal loops due to fecal retention.

The animal presented pendulous abdomen and abdominal dilatation due to rectal stenosis.

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5.59 Rectal stenosis in a growing pig. Note intense narrowing of the lumen due to sclerosis of the intestinal wall. Usually associated with ulcerative proctitis and thrombosis of hemorrhoid vessels as sequel of salmonellosis. However, stenosis is also observed in rectal prolapses without antecedents of enterocolitis by Salmonella.

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6.45 Pyometra in a sow presenting vulvo-vaginal exudates. The animal died suddenly. Note dilatation of the uterine horns, bloodish and purulent content, hyperplasia and necrosis of endometrium and severe edematization of the wall of the uterus. Pyometra by Staphylococcus sp. and E. coli.

6.46 Detailed section of anterior uterus presenting intense edematization of the wall.

The characteristics of E. coli identified corresponded with an endotoxic strain.

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6.47 Pyometra in a sow suffering from “dirty sow syndrome�. Note uterus dilatation, abundance of pus and swollen endometrium. Mixed infection Staphylococcus sp. and Streptococcus sp.

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6.45 Pyometra in a sow presenting vulvo-vaginal exudates. The animal died suddenly. Note dilatation of the uterine horns, bloodish and purulent content, hyperplasia and necrosis of endometrium and severe edematization of the wall of the uterus. Pyometra by Staphylococcus sp. and E. coli.

6.46 Detailed section of anterior uterus presenting intense edematization of the wall.

The characteristics of E. coli identified corresponded with an endotoxic strain.

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6.47 Pyometra in a sow suffering from “dirty sow syndrome�. Note uterus dilatation, abundance of pus and swollen endometrium. Mixed infection Staphylococcus sp. and Streptococcus sp.

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