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Liver and glutathione diseases

Quicksilver Scientific 5/17/2018


Glutathione is a small protein produced inside the cells that have important functions in the organism, so vital that without the glutathione it would simply be impossible our survival. A fall in glutathione levels represents a determinant factor in liver damage, since glutathione is found mainly in this organ in which case it works as a protector of oxidative stress (cellular oxidation) and toxic (Eliminandolos organism).

A study of liver and glutathione diseases In a small study, abnormally low concentrations of glutathione in blood were observed in patients with cirrhosis, independence of their diet. A larger study showed a significant decrease in blood glutathione of 48 patients with cirrhosis against 18 healthy volunteers. A significant drop in cytokine levels was also observed in severe cirrhosis. Researchers measured the levels of glutathione and oxidized glutathione (GSSG), in patients with chronic alcoholism, others with liver diseases not related to alcohol consumption (fatty liver, chronic and acute hepatitis, cirrhosis), and control patients No liver disease.


They found that glutathione was at low levels in alcoholic and nonalcoholic patients with liver diseases, compared with the control group. The level of the oxidized glutathione (produced when the glutathione is used) was much higher in these groups, indicating a high level of oxidative stress in the liver. The investigators postulated that the decrease in glutathione and the increase in GSSG contributes to the damage of the liver and to a toxic risk in these patients, while the fundamental cellular function is altered like the synthesis of proteins, historical enzymatic, Transport processes, and mechanisms of growth. Other studies have also documented a decrease in blood and liver glutathione in patients with acute viral hepatitis, and in chronic cases of hepatitis, liver disease from alcoholism, and non-alcoholic cirrhosis. Glutathione deficiency caused by a toxin can lead to the liver being more vulnerable to other toxins. An example is the taking of acetaminophen imposed on liver damaged by alcoholism. In a group of alcoholics chronic with glutathione deficiency, showed abnormally low levels of glutathione in blood after the taking of acetaminophen, and are therefore more predipuestos to damage liver larger by other toxic agents.

Glutathione deficiency in Liver diseases Glutathione is a small protein produced inside cells that have important functions in the organism, so vital that without glutathione it would simply be impossible our survival. A drop in glutathione levels represents a determinant factor in liver damage, as glutathione is found mainly in this organ in which case it works as a protector of oxidative stress (cellular oxidation) and toxic (Eliminandolos organism).


In a small study, abnormally low concentrations of glutathione in blood were observed in patients with cirrhosis, independence of their diet. A larger study showed a significant decrease in the blood glutathione of 48 patients with cirrhosis against 18 healthy volunteers. A significant drop in cytokine levels was also observed in severe cirrhosis. Researchers measured levels of glutathione and oxidized glutathione (GSSG), in patients with chronic alcoholism, others with liver diseases not related to alcohol consumption (fatty liver, acute and chronic hepatitis, cirrhosis), and control patients No liver disease. They found that glutathione was at low levels in Alcoholics and nonalcoholic patients with liver diseases, compared with the control group. The level of oxidized glutathione (produced when glutathione is used) was much higher in these groups, indicating a high level of oxidative stress in the liver. The researchers postulated that the decrease in glutathione and/or the increase in GSSG contributes to the damage of the liver and to a toxic risk in these patients, while altering the fundamental cellular function like the synthesis of proteins, enzymatic historical, Transport processes, and secretion mechanisms.


Other studies have also documented a decrease in glutathione in blood and liver in patients with acute viral hepatitis, and in chronic cases of hepatitis, liver disease by alcoholism, and non-alcoholic cirrhosis. The deficiency of glutathione caused by a toxin can lead to the liver to be more vulnerable to other toxins. An example is the taking of acetaminophen imposed on liver damaged by alcoholism. In a group of chronic alcoholics with glutathione deficiency, they manifested abnormally low levels of glutathione in the blood after the taking of acetaminophen, and are consequently more predipuestos to greater hepatic damage by other toxic agents.

Liver, hepatitis and glutathione Hepatitis is an inflammatory disease of the liver. The 2 most important types of hepatitis are toxic or alcoholic hepatitis and infectious hepatitis, usually caused by type A, B, and C viruses. Toxic Hepatitis. This is a non-infectious condition caused by exposure to liver-damaging chemicals. Liver-damaging agents are many, but alcohol abuse is the culprit of most cases. Alcoholism is a chronic disease and prolonged inflammation of the liver leads to what is known as hepatic cirrhosis.

Infectious Hepatitis. Infectious hepatitis is the most common form of all serious infectious disease. It is estimated that only in the USA half a million people get this disease. Given the growth of a relatively new type of hepatitis virus, the C virus, this number can be increased gradually over the next few years.


The evolution of the disease is variable; it can go from showing no symptoms to causing death in a small percentage of cases. Most people with infectious hepatitis suffer from a few weeks of flu-like symptoms, consisting of fatigue, pain, average fever, loss of appetite, abdominal pain, nausea, and vomiting. More severe cases show dark-colored urine, jaundice (yellow skin), itching, very clear stools, and altered mental states. The progression of inflammation in the liver determines how well the liver will work. When there is liver dysfunction the liver will be unable to filter and eliminate toxins, regulate digestion, and regulate the composition of chemicals in blood, process and store nutrients, and other vital functions. The extent of this damage can be measured with liver function tests (CFT), a measure of certain liver enzymes in the blood.


Chronic Hepatitis. Some cases can happen to become a chronic hepatitis, which is a much more serious problem. In these cases of toxic hepatitis patients should be released from the toxin, a major challenge in the case of alcohol.

Prevention. Prevention is the best way to deal with all forms of hepatitis. Things like: To have a correct hygiene, not to use products derived from the blood, vaccination, not to use toxic like alcohol and avoiding the contact of fluids with sick people.

The glutathione and the liver. The Hematologist knows that glutathione has an extremely important role in the functioning of the liver, being the organ with the highest amount of glutathione in the organism. This is because it works as a key filter for the detoxification of the organism. In Phase 1 of the detoxification process, the liver transforms these toxins into water-soluble substances. In Phase 2, Glutathione helps neutralize these products and helps the body eliminate them through the intestines or kidneys. If these two phases of detoxification fail for any reason, the toxins accumulate in the body, producing a disease. Science

has

known

for

many

years

that

glutathione

deficiency

invariably accompanies liver damage. When hepatitis comes from an intoxication of medications such as acetaminophen (Tylenol, Parasol, etc.) Some precursor of glutathione should be used to retexture the levels. There is much research that has promoted the use of products to raise glutathione levels in these cases. This approach has even been used for the treatment of culminant hepatic failure.


Many patients with hepatitis due to alcoholism have been treated with great success by elevating the levels of glutathione.

Glutathione in the treatment of infectious Hepatitis. It was demonstrated in the Max Planck Institute the antiviral properties of glutathione precursors in improving liver functions, as well as the immunological parameters in type B Hepatitis. These improvements continued even after the treatment was completed, reflecting the long-term benefits of this approach. Treatments for Hepatitis C are far from ideal. A team of Italian scientists demonstrated a systematic drop in glutathione levels in patients of this type, making it resistant to the virus and its treatments, such as Interferon. And on the contrary, elevating glutathione levels showed that Interferon had a more positive effect. The liver is one of the most complex and large organs of our organism. It is closely linked to a number of factors that determine overall health. Glutathione is a key element in the proper functioning of the liver. Low levels of glutathione only because worsening of the disease, raising the levels protects us from these health problems.


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Liver and glutathione diseases  

Glutathione is a small protein produced inside the cells that have important functions in the organism, so vital that without the glutathion...

Liver and glutathione diseases  

Glutathione is a small protein produced inside the cells that have important functions in the organism, so vital that without the glutathion...

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