The Healthy Geezer with Fred Cietti When to Push Yourself Away From The Table
. If you want to control your weight, is it better to eat three meals a day, or should you eat more, smaller meals?
. There is no scientifically proven
answer to this question. Until there is, I’d suggest simply reducing your total food intake for each day until your weight goes down. [Personal note: I’ve struggled to keep pounds off all my life. I’ve learned that calories count. You take in more than you burn; you gain weight. You burn more than you eat; you lose weight. Exercise helps, but the calorie burn-off usually doesn’t amount to much. The best exercise is pushing yourself away from the table.] Eating a bunch of small meals a day instead of breakfast-lunch-dinner is part of the popular media right now, which means you should be hearing “fad alert” in your head. Here are some of the claims: • The body burns calories to digest. Eating six to eight meals a day enables your body to use more calories to aid digestion. • Eating lots of meals rather than three will boost metabolism and control blood sugar. • More meals means less stored fat in the body. • When people consume the same number of calories in a single daily meal rather than three, they show significant increases in blood pressure, total cholesterol levels and levels of bad LDL cholesterol. • Eating every three-to-four hours can ward off hunger and prevent binges that lead to weight gain. • Eating more often helps regulate proper digestion to prevent gastrointestinal problems. • Eight meals a day will increase energy levels and accelerate muscle growth. To repeat, there is no proof that eating more frequently does any of the above. My own conclusion about weight control was confirmed in an editorial that appeared in the American Journal of Clinical Nutrition. A team of nutrition researchers concluded that weight loss comes down to “how much energy (or calories) is consumed as opposed to how often or how regularly one eats.” However, there was a recent study that indicated we may be better off eating only three meals a day. The study was done on mice, so the findings have to be confirmed by tests on humans. Satchidananda Panda, a regulatory biologist at the Salk Institute in La Jolla, led the study published by the journal Cell Metabolism. Panda and his team put groups of mice on different eating regimens for 100 days. Mice in two of the groups ate high-fat, high-calorie food. One of these groups was allowed to snack throughout the night and day. The other group had access to the calorieloaded food only for eight hours at night, when they were most active. A control group of mice ate normal food, not the high-calorie food. The mice that ate only for eight hours were almost as lean as mice in a control group that ate regular food. But the mice that ate around the clock became obese, even though they consumed the same amount of fat and calories as their counterparts on the time-restricted diet.
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PAGE FIVE • MARCH 2-8, 2017
Living with MS with Dee Dean Inflammatory Disease Trigger Discovered
nstitute scientists have revealed a potent inflammatory molecule released by dying cells triggers inflammation during necroptosis, a recently described form of cell death linked to inflammatory disease. The discovery could lead to new and existing medicines that target the molecule being investigated as a way of treating inflammatory diseases, such as psoriasis and inflammatory bowel disease. Dr Lisa Lindqvist, Dr Kate Lawlor, Dr James Vince and PhD student Ms Stephanie Conos led research that showed interleukin-1 beta (IL-1) triggers inflammation during necroptotic cell death. Necroptosis is important for protecting us against infections, by sacrificing infected or diseased cells ‘for the greater good’. However, necroptosis can become inappropriately or excessively activated, triggering damaging inflammation that leads to inflammatory disease.
Dr Lindqvist said the discovery challenged a long-standing dogma that inflammation triggered by necroptosis was a byproduct of dead cell debris. “Our research has pinpointed that, during necroptosis, dying cells release IL-1, a potent inflammatory signal,” Dr Lindqvist said. “Now that we have discovered IL-1 is the ‘root’ of the inflammation associated with necroptosis, we speculate that targeting this molecule could be an effective way of treating inflammatory diseases.”
Future treatments The findings suggest that targeting IL-1 could suppress inflammation associated with multiple inflammatory diseases, including Multiple Sclerosis, ischemia-reperfusion injury, atherosclerosis, liver disease, pancreatitis, psoriasis, inflammatory bowel disease, and infectious diseases. “Our research suggests
that existing drugs that block IL-1 might be useful in treating these diseases,” Dr Lindqvist said. “We are also exploring how IL-1 is signalled to be secreted during necroptosis, so that we can create new drugs to stop its release and reduce inflammation to treat inflammatory diseases.”
Source: Walter Reed Army Institute of Research
Dean has been fighting Multiple Sclerosis for 30 years. She continually studies and researches the disease to educate herself. She writes this column as a community service to share her findings and to raise public awareness about MS. The opinions and experiences shared are her own. Dean is NOT a medical doctor. ALWAYS check with your doctor first before trying a new therapy. This column is intended for informational purposes only. Dean can be reached at firstname.lastname@example.org. NOTE: Dean is the recipient of the 2004 STAR Community Outreach Award by the MS Society Dec. 2, 2004, the American Red Cross Real Hero Wendell Cutting Humanitarian Award, Oct. 13, 2006 , the Stoney Community Service Award, February 29, 2008, Women in Leadership Award for Art/Media/ Culture Oct. 29, 2010, El Cajon Citizen of The Year Nominee Feb. 2013 and 2017 and Recipient of the National MS Society’s 2014 Media Partner of The Year, Feb. 10, 2015.
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